Best adjuvant (assist) for chemotherapy | 1+1>487% |
Effectively improve chemotherapy effect, treatment, immunity |
Reduce side effects and recurrence |
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work |
Abstract / Summary / Overview of Apoptosis
Why do cells undergo apoptosis?
The relationship between cancer cells and apoptosis
Where are the weaknesses and symptoms of cancer cells?
Are cancer cells aggressive?
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
Solamargine vs cancer
Best Chemotherapy Adjuvant (1+1>478%) Effectively improve chemotherapy effect and cure
Best adjuvant (assist) for chemotherapy | 1+1>487%
Effectively improve chemotherapy effect, treatment, immunity
Reduce side effects and recurrence
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work
Abstract / Summary / Overview of Apoptosis
Overview of apoptosis
•Programmed cell death
•Apoptosis is a form of programmed cell death, or “cellular suicide.”
•Apoptosis is different from necrosis, in which cells die due to injury.
•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.
Why do cells undergo apoptosis?
- Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
- Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
- Apoptosis is part of development
- In many organisms, programmed cell death is a normal part of development.
The relationship between cancer cells and apoptosis
Apoptosis can eliminate infected or cancerous cells.
When a cell’s DNA is damaged, it will typically detect the damage and try to repair it.
If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA.
When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.
However, “successful” cancer cells successfully evade the process of apoptosis.
This allows them to divide out of control and accumulate mutations (changes in their DNA).
Apoptosis is key to immune function
Apoptosis also plays an essential role in the development and maintenance of a healthy immune system.
Where are the weaknesses and symptoms of cancer cells?
The symptoms of cancer cells are in the nucleus.
The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.
DNA mutations also mutate in the nucleus.
Therefore, to treat cancer cells, we must first enter the nucleus.
Let the “regulatory cell gene” mechanism enter the nucleus to regulate
Are cancer cells aggressive?
After the action of Solamargine, the aggressiveness of cancer cells is alleviated.
So after using Solamargine, many patients feel that I am half better.
Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
When Solamargine enter,
Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.
Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.
Reduced drug resistance
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.
Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?
Solamargine vs cancer
Solamargine vs cancer
The picture shows the death of cancer cells.
The black and black parts are cancer cell nuclei.
Even if the nucleus ruptures, the cancer cells will die.
The figure shows that cancer cells can cause death.
The figure shows that cancer cells can cause death.
The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.
The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.
The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.
Best Chemotherapy Adjuvant (1+1>487%) Effectively improve chemotherapy effect and treatment
ANTI-CANCER
Patent protection in 32 nations.
A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.
A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells
SR-T100 combination therapy with effective result against breast cancer cells.
Combination Therapy | Research results for lung cancer cells.
A. Chemotherapy (100μM), 16% of cancer cell apoptosis.
B. Alone SM (4.8μM), 28% of cancer cell apoptosis.
C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.
D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.
SM has a clearing effect better than Chemotherapy.
The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.
SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).
SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times)
Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004
Solamargine Q&A (English)
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Rétinoblastome | taux de survie | Contents
Rétinoblastome | taux de survie | Etymology and definitions
Rétinoblastome | taux de survie | Signs and symptoms
Rétinoblastome | taux de survie | Local symptoms
Rétinoblastome | taux de survie | Systemic symptoms
Rétinoblastome | taux de survie | Metastasis
Rétinoblastome | taux de survie | Causes
Rétinoblastome | taux de survie | Chemicals
Rétinoblastome | taux de survie | Diet and exercise
Rétinoblastome | taux de survie | Infection
Rétinoblastome | taux de survie | Radiation
Rétinoblastome | taux de survie | Heredity
Rétinoblastome | taux de survie | Physical agents
Rétinoblastome | taux de survie | Hormones
Rétinoblastome | taux de survie | Autoimmune diseases
Rétinoblastome | taux de survie | Pathophysiology
Rétinoblastome | taux de survie | Genetics
Rétinoblastome | taux de survie | Epigenetics
Rétinoblastome | taux de survie | Metastasis
Rétinoblastome | taux de survie | Metabolism
Rétinoblastome | taux de survie | Diagnosis
Rétinoblastome | taux de survie | Classification
Rétinoblastome | taux de survie | Prevention
Rétinoblastome | taux de survie | Dietary
Rétinoblastome | taux de survie | Medication
Rétinoblastome | taux de survie | Vaccination
Rétinoblastome | taux de survie | Screening
Rétinoblastome | taux de survie | Recommendations
Rétinoblastome | taux de survie | Genetic testing
Rétinoblastome | taux de survie | Management
Rétinoblastome | taux de survie | Chemotherapy
Rétinoblastome | taux de survie | Radiation
Rétinoblastome | taux de survie | Surgery
Rétinoblastome | taux de survie | Palliative care
Rétinoblastome | taux de survie | Immunotherapy
Rétinoblastome | taux de survie | Laser therapy
Rétinoblastome | taux de survie | Alternative medicine
Rétinoblastome | taux de survie | Prognosis
Rétinoblastome | taux de survie | Epidemiology
Rétinoblastome | taux de survie | History
Rétinoblastome | taux de survie | Society and culture
Rétinoblastome | taux de survie | Economic effect
Rétinoblastome | taux de survie | Workplace
Rétinoblastome | taux de survie | Research
Rétinoblastome | taux de survie | Pregnancy
Rétinoblastome | taux de survie | Other animals
Rétinoblastome | taux de survie | Notes
Rétinoblastome | taux de survie | Further reading
Rétinoblastome | taux de survie | External links