Uterine Sarcoma | Effectively improve the cure rate. | Increase survival rate | Stage | Stage 0 | Stage 1 | Stage 2 | Stage 3 | Stage 4

Uterine Sarcoma | Effectively improve the cure rate. | Increase survival rate | Stage | Stage 0 | Stage 1 | Stage 2 | Stage 3 | Stage 4

Best adjuvant (assist) for chemotherapy | 1+1>487% |

Effectively improve chemotherapy effect, treatment, immunity. 

Reduce side effects and recurrence.  

Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work | 

Abstract / Summary / Overview of Apoptosis

Why do cells undergo apoptosis?

The relationship between cancer cells and apoptosis.

Where are the weaknesses and symptoms of cancer cells?

Are cancer cells aggressive?

Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)

Solamargine's major function mechanism:

Solamargine vs cancer

Best Chemotherapy Adjuvant  (1+1>478%) 

Effectively improve chemotherapy effect and cure.

When cancer cells are less resistant to drugs, chemotherapy becomes more effective.



Extract : https://www.cancer.gov/types/uterine/patient/uterine-sarcoma-treatment-pdq

Uterine Sarcoma Treatment

General Information About Uterine Sarcoma

KEY POINTS

  • Uterine sarcoma is a disease in which malignant (cancer) cells form in the muscles of the uterus or other tissues that support the uterus.
  • Past treatment with radiation therapy to the pelvis can increase the risk of uterine sarcoma.
  • Signs of uterine sarcoma include abnormal bleeding.
  • Tests that examine the uterus are used to diagnose uterine sarcoma.
  • Certain factors affect prognosis (chance of recovery) and treatment options.



Abstract / Summary / Overview of Apoptosis

Apoptosis.jpg

Overview of apoptosis

•Programmed cell death.

•Apoptosis is a form of programmed cell death, or “cellular suicide.”

•Apoptosis is different from necrosis, in which cells die due to injury.

•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.


Why do cells undergo apoptosis?

  • Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
  • Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
  • Apoptosis is part of development.
  • In many organisms, programmed cell death is a normal part of development.


The relationship between cancer cells and apoptosis

Apoptosis can eliminate infected or cancerous cells.

When a cell’s DNA is damaged, it will typically detect the damage and try to repair it. 

If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA. 

When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.

However, “successful” cancer cells successfully evade the process of apoptosis.

This allows them to divide out of control and accumulate mutations (changes in their DNA).

Apoptosis is key to immune function.

Apoptosis also plays an essential role in the development and maintenance of a healthy immune system. 


Where are the weaknesses and symptoms of cancer cells?

The symptoms of cancer cells are in the nucleus.

The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.

DNA mutations also mutate in the nucleus.

Therefore, to treat cancer cells, we must first enter the nucleus.

Let the “regulatory cell gene” mechanism enter the nucleus to regulate.


Are cancer cells aggressive?

After the action of Solamargine, the aggressiveness of cancer cells is alleviated.

So after using Solamargine, many patients feel that I am half better.

Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.



Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)

sr-t100_apoptosis_mechanism005.jpg


Solamargine's major function mechanism:

When Solamargine enter,

Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.

Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.

Reduced drug resistance.

When cancer cells are less resistant to drugs, chemotherapy becomes more effective.

Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.


Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?

Chemotherapy_01.jpg



Solamargine vs cancer

cell apoptosis.jpg

Solamargine vs cancer

The picture shows the death of cancer cells.

The black and black parts are cancer cell nuclei.

Even if the nucleus ruptures, the cancer cells will die.

The figure shows that cancer cells can cause death. 

cancer cell apoptosis_01_800.jpg

The figure shows that cancer cells can cause death.

The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.

The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.

The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.



Best Chemotherapy Adjuvant (1+1>487%) 

Effectively improve chemotherapy effect and treatment.solamargine vs cancer_lung cancer cell.jpg

ANTI-CANCER 

Patent protection in 32 nations. 

A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.

solamargine vs cancer_breast cancer cell_01_800.jpgA comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells.

solamargine combined treatment therapy_01_800.jpg

SR-T100 combination therapy with effective result against breast cancer cells.


solamargine combined treatment therapy_03R12_800.jpg

Combination Therapy   |   Research results for lung cancer cells. 

A. Chemotherapy    (100μM), 16% of cancer cell apoptosis. 

B. Alone SM (4.8μM), 28% of cancer cell apoptosis. 

C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.  

D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.  

SM has a clearing effect better than Chemotherapy. 

The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.  

SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).  

SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times).  

Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004.


justnow_02.jpg

The best solution for cancer cells

Solamargine Q&A (English)



Cancer Medical Care | Side Effects of Chemotherapy | Improve Anemia | Improve Low Red Blood Cells | Improve Low Hemoglobin (Decrease/Decrease/Decrease/Insufficiency) 

Cancer Medical Care | Chemotherapy by-products | Leukopenia | Neutropenia 

Cancer Medical Care | Chemotherapy Side Effects| Improve Bleeding| Improve Platelet Decrease| Improve Platelet Deficiency| Improve Platelet Low| Improve Thrombocytopenia 

Cancer Medical Care | Six indicators of physical health self-assessment: sleep, appetite, excretion, physical strength, mental strength, and psychology (emotion, mind) 
 


Chemotherapy | Increase cure rate 
Reduce cancer recurrence 
Apoptotic (Correct/ Guide) bad behavior 
Cure all diseases | Panacea | Package health 
How to Longevity? 
Want to Change? 
Life Above All | Release life is best for oneself (me / yourself) 
Eliminate misfortune! 
Good affinities with others. 
Beauty in Nature



survival rate | cancer | Contents
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survival rate | cancer | Other animals
survival rate | cancer | Notes
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Uterine Sarcoma | survival rate | Contents
Uterine Sarcoma | survival rate | Etymology and definitions
Uterine Sarcoma | survival rate | Signs and symptoms
Uterine Sarcoma | survival rate | Local symptoms
Uterine Sarcoma | survival rate | Systemic symptoms
Uterine Sarcoma | survival rate | Metastasis
Uterine Sarcoma | survival rate | Causes
Uterine Sarcoma | survival rate | Chemicals
Uterine Sarcoma | survival rate | Diet and exercise
Uterine Sarcoma | survival rate | Infection
Uterine Sarcoma | survival rate | Radiation
Uterine Sarcoma | survival rate | Heredity
Uterine Sarcoma | survival rate | Physical agents
Uterine Sarcoma | survival rate | Hormones
Uterine Sarcoma | survival rate | Autoimmune diseases
Uterine Sarcoma | survival rate | Pathophysiology
Uterine Sarcoma | survival rate | Genetics
Uterine Sarcoma | survival rate | Epigenetics
Uterine Sarcoma | survival rate | Metastasis
Uterine Sarcoma | survival rate | Metabolism
Uterine Sarcoma | survival rate | Diagnosis
Uterine Sarcoma | survival rate | Classification
Uterine Sarcoma | survival rate | Prevention
Uterine Sarcoma | survival rate | Dietary
Uterine Sarcoma | survival rate | Medication
Uterine Sarcoma | survival rate | Vaccination
Uterine Sarcoma | survival rate | Screening
Uterine Sarcoma | survival rate | Recommendations
Uterine Sarcoma | survival rate | Genetic testing
Uterine Sarcoma | survival rate | Management
Uterine Sarcoma | survival rate | Chemotherapy
Uterine Sarcoma | survival rate | Radiation
Uterine Sarcoma | survival rate | Surgery
Uterine Sarcoma | survival rate | Palliative care
Uterine Sarcoma | survival rate | Immunotherapy
Uterine Sarcoma | survival rate | Laser therapy
Uterine Sarcoma | survival rate | Alternative medicine
Uterine Sarcoma | survival rate | Prognosis
Uterine Sarcoma | survival rate | Epidemiology
Uterine Sarcoma | survival rate | History
Uterine Sarcoma | survival rate | Society and culture
Uterine Sarcoma | survival rate | Economic effect
Uterine Sarcoma | survival rate | Workplace
Uterine Sarcoma | survival rate | Research
Uterine Sarcoma | survival rate | Pregnancy
Uterine Sarcoma | survival rate | Other animals
Uterine Sarcoma | survival rate | Notes
Uterine Sarcoma | survival rate | Further reading
Uterine Sarcoma | survival rate | External links




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