Multiple endocrine neoplasia syndrome | Where are the weaknesses and symptoms of cancer cells? | Increase survival rate | Stage | Stage 0 | Stage 1 | Stage 2 | Stage 3 | Stage 4

Multiple endocrine neoplasia syndrome | Where are the weaknesses and symptoms of cancer cells? | Increase survival rate | Stage | Stage 0 | Stage 1 | Stage 2 | Stage 3 | Stage 4

Best adjuvant (assist) for chemotherapy | 1+1>487% |

Effectively improve chemotherapy effect, treatment, immunity. 

Reduce side effects and recurrence.  

Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work | 

Abstract / Summary / Overview of Apoptosis.

Why do cells undergo apoptosis?

The relationship between cancer cells and apoptosis.

Where are the weaknesses and symptoms of cancer cells?

Are cancer cells aggressive?

Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)

Solamargine's major function mechanism:

Solamargine vs cancer

Best Chemotherapy Adjuvant. (1+1>478%) 

Effectively improve chemotherapy effect and cure.

When cancer cells are less resistant to drugs, chemotherapy becomes more effective.



Extract : https://www.cancer.gov/types/multiple-endocrine-neoplasia/patient-child-men-syndromes-treatment-pdq

Childhood Multiple Endocrine Neoplasia (MEN) Syndromes Treatment

General Information About Childhood Multiple Endocrine Neoplasia (MEN) Syndromes

KEY POINTS

  • Multiple endocrine neoplasia (MEN) syndromes are inherited disorders that affect the endocrine system.
  • There are several types of MEN syndromes and each type may cause different conditions or cancers.
  • MEN1 syndrome usually causes tumors in the parathyroid gland, pituitary gland, or islet cells of the pancreas.
  • MEN2A syndrome may cause medullary thyroid cancer, pheochromocytoma, or parathyroid gland disease.
  • MEN2B syndrome causes several conditions.
  • Children with MEN2A syndrome, MEN2B syndrome, or FMTC may need genetic testing.
  • Tests used to diagnose and stage cancers related to MEN syndromes depend on the signs and symptoms and the patient's family history.
  • If cancer, such as medullary thyroid cancer, has formed, tests are done to find out if cancer cells have spread to nearby areas or to other parts of the body.
  • There are three ways that cancer spreads in the body.
  • Cancer may spread from where it began to other parts of the body. 



Abstract / Summary / Overview of Apoptosis. 

Apoptosis.jpg

Overview of apoptosis

•Programmed cell death.

•Apoptosis is a form of programmed cell death, or “cellular suicide.”

•Apoptosis is different from necrosis, in which cells die due to injury.

•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.


Why do cells undergo apoptosis?

  • Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
  • Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
  • Apoptosis is part of development.
  • In many organisms, programmed cell death is a normal part of development.


The relationship between cancer cells and apoptosis.

Apoptosis can eliminate infected or cancerous cells.

When a cell’s DNA is damaged, it will typically detect the damage and try to repair it. 

If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA. 

When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.

However, “successful” cancer cells successfully evade the process of apoptosis.

This allows them to divide out of control and accumulate mutations (changes in their DNA).

Apoptosis is key to immune function.

Apoptosis also plays an essential role in the development and maintenance of a healthy immune system. 


Where are the weaknesses and symptoms of cancer cells?

The symptoms of cancer cells are in the nucleus.

The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.

DNA mutations also mutate in the nucleus.

Therefore, to treat cancer cells, we must first enter the nucleus.

Let the “regulatory cell gene” mechanism enter the nucleus to regulate.


Are cancer cells aggressive?

After the action of Solamargine, the aggressiveness of cancer cells is alleviated.

So after using Solamargine, many patients feel that I am half better.

Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.



Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work). 

sr-t100_apoptosis_mechanism005.jpg


Solamargine's major function mechanism:

When Solamargine enter,

Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.

Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.

Reduced drug resistance.

When cancer cells are less resistant to drugs, chemotherapy becomes more effective.

Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.


Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?

Chemotherapy_01.jpg



Solamargine vs cancer

cell apoptosis.jpg

Solamargine vs cancer

The picture shows the death of cancer cells.

The black and black parts are cancer cell nuclei.

Even if the nucleus ruptures, the cancer cells will die.

The figure shows that cancer cells can cause death. 

cancer cell apoptosis_01_800.jpg

The figure shows that cancer cells can cause death.

The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.

The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.

The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.



Best Chemotherapy Adjuvant. (1+1>487%) 

Effectively improve chemotherapy effect and treatment.solamargine vs cancer_lung cancer cell.jpg

ANTI-CANCER 

Patent protection in 32 nations. 

A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.

solamargine vs cancer_breast cancer cell_01_800.jpgA comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells.

solamargine combined treatment therapy_01_800.jpg

SR-T100 combination therapy with effective result against breast cancer cells.


solamargine combined treatment therapy_03R12_800.jpg

Combination Therapy   |   Research results for lung cancer cells. 

A. Chemotherapy    (100μM), 16% of cancer cell apoptosis. 

B. Alone SM (4.8μM), 28% of cancer cell apoptosis. 

C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.  

D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.  

SM has a clearing effect better than Chemotherapy. 

The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.  

SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).  

SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times).  

Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004.


justnow_02.jpg

The best solution for cancer cells.

Solamargine Q&A (English)



Cancer Medical Care | Side Effects of Chemotherapy | Improve Anemia | Improve Low Red Blood Cells | Improve Low Hemoglobin (Decrease/Decrease/Decrease/Insufficiency) 

Cancer Medical Care | Chemotherapy by-products | Leukopenia | Neutropenia 

Cancer Medical Care | Chemotherapy Side Effects | Improve Bleeding | Improve Platelet Decrease | Improve Platelet Deficiency | Improve Platelet Low | Improve Thrombocytopenia 

Cancer Medical Care | Six indicators of physical health self-assessment: sleep, appetite, excretion, physical strength, mental strength, and psychology (emotion, mind) 
 


Chemotherapy | Increase cure rate 
Reduce cancer recurrence 
Apoptotic (Correct/ Guide) bad behavior 
Cure all diseases | Panacea | Package health 
How to Longevity? 
Want to Change? 
Life Above All | Release life is best for oneself (me / yourself) 
Eliminate misfortune! 
Good affinities with others. 
Beauty in Nature



survival rate | cancer | Contents
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Multiple endocrine neoplasia syndrome | survival rate | Contents
Multiple endocrine neoplasia syndrome | survival rate | Etymology and definitions
Multiple endocrine neoplasia syndrome | survival rate | Signs and symptoms
Multiple endocrine neoplasia syndrome | survival rate | Local symptoms
Multiple endocrine neoplasia syndrome | survival rate | Systemic symptoms
Multiple endocrine neoplasia syndrome | survival rate | Metastasis
Multiple endocrine neoplasia syndrome | survival rate | Causes
Multiple endocrine neoplasia syndrome | survival rate | Chemicals
Multiple endocrine neoplasia syndrome | survival rate | Diet and exercise
Multiple endocrine neoplasia syndrome | survival rate | Infection
Multiple endocrine neoplasia syndrome | survival rate | Radiation
Multiple endocrine neoplasia syndrome | survival rate | Heredity
Multiple endocrine neoplasia syndrome | survival rate | Physical agents
Multiple endocrine neoplasia syndrome | survival rate | Hormones
Multiple endocrine neoplasia syndrome | survival rate | Autoimmune diseases
Multiple endocrine neoplasia syndrome | survival rate | Pathophysiology
Multiple endocrine neoplasia syndrome | survival rate | Genetics
Multiple endocrine neoplasia syndrome | survival rate | Epigenetics
Multiple endocrine neoplasia syndrome | survival rate | Metastasis
Multiple endocrine neoplasia syndrome | survival rate | Metabolism
Multiple endocrine neoplasia syndrome | survival rate | Diagnosis
Multiple endocrine neoplasia syndrome | survival rate | Classification
Multiple endocrine neoplasia syndrome | survival rate | Prevention
Multiple endocrine neoplasia syndrome | survival rate | Dietary
Multiple endocrine neoplasia syndrome | survival rate | Medication
Multiple endocrine neoplasia syndrome | survival rate | Vaccination
Multiple endocrine neoplasia syndrome | survival rate | Screening
Multiple endocrine neoplasia syndrome | survival rate | Recommendations
Multiple endocrine neoplasia syndrome | survival rate | Genetic testing
Multiple endocrine neoplasia syndrome | survival rate | Management
Multiple endocrine neoplasia syndrome | survival rate | Chemotherapy
Multiple endocrine neoplasia syndrome | survival rate | Radiation
Multiple endocrine neoplasia syndrome | survival rate | Surgery
Multiple endocrine neoplasia syndrome | survival rate | Palliative care
Multiple endocrine neoplasia syndrome | survival rate | Immunotherapy
Multiple endocrine neoplasia syndrome | survival rate | Laser therapy
Multiple endocrine neoplasia syndrome | survival rate | Alternative medicine
Multiple endocrine neoplasia syndrome | survival rate | Prognosis
Multiple endocrine neoplasia syndrome | survival rate | Epidemiology
Multiple endocrine neoplasia syndrome | survival rate | History
Multiple endocrine neoplasia syndrome | survival rate | Society and culture
Multiple endocrine neoplasia syndrome | survival rate | Economic effect
Multiple endocrine neoplasia syndrome | survival rate | Workplace
Multiple endocrine neoplasia syndrome | survival rate | Research
Multiple endocrine neoplasia syndrome | survival rate | Pregnancy
Multiple endocrine neoplasia syndrome | survival rate | Other animals
Multiple endocrine neoplasia syndrome | survival rate | Notes
Multiple endocrine neoplasia syndrome | survival rate | Further reading
Multiple endocrine neoplasia syndrome | survival rate | External links




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