Best adjuvant (assist) for chemotherapy | 1+1>487% |
Effectively improve chemotherapy effect, treatment, immunity.
Reduce side effects and recurrence.
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work |
Abstract / Summary / Overview of Apoptosis.
Why do cells undergo apoptosis?
The relationship between cancer cells and apoptosis.
Where are the weaknesses and symptoms of cancer cells?
Are cancer cells aggressive?
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
Solamargine vs cancer
Best Chemotherapy Adjuvant. (1+1>478%)
Effectively improve chemotherapy effect and cure.
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Extract : https://cancer.ca/en/cancer-information/cancer-types/brain-and-spinal-cord-childhood/what-is-a-childhood-brain-or-spinal-tumour/optic-and-hypothalamic-gliomas
Treatment of optic and hypothalamic gliomas
The main goal of treatment is to save the child’s sight and hormone functions. Treatment for optic and hypothalamic gliomas depends on the:
- child’s age and general health
- location and size of the tumour
- extent of vision loss or other symptoms
Children with neurofibromatosis often have more slow-growing tumours. If the tumour is not growing, it may be left untreated and checked regularly with MRIs.
Treatment will be given if the tumour is large, if the tumour is growing, if there is a threat to the child’s sight or if symptoms are getting worse.
Children younger than 1 often have a more aggressive tumour and require more urgent treatment.
Surgery
Surgery is only used if the tumour is easily accessible and can be safely removed or if symptoms are getting worse. Surgery for optic and hypothalamic tumours is difficult as the tumours are often difficult to reach. They often invade the nerve leading to the eye, and surgery can result in the loss of vision in one or both eyes. Surgery may also be used to relieve a buildup of cerebrospinal fluid (CSF).
Radiation therapy
Radiation therapy is the main treatment when surgery cannot be done and treatment is needed. It can shrink the tumour or slow its growth. In children under 3 years old, radiation therapy can be damaging to developing brain cells. In these cases, chemotherapy may be given first, followed by radiation therapy, if needed, once the child is over the age of 3.
Chemotherapy
Chemotherapy has shown some success in treating these tumours. It can shrink the tumour and delay the need for radiation in young children. Chemotherapy treatment usually lasts for a year or longer. It is often given as an outpatient treatment, which means that your child will not have to stay overnight. The most common chemotherapy drugs used are vincristine (Oncovin) and carboplatin (Paraplatin, Paraplatin AQ).
Active surveillance
Active surveillance may be an option if the tumour is not growing and there are no symptoms. The child will be watched closely using MRIs for any signs of tumour growth. Treatment is started when the tumour progresses.
Abstract / Summary / Overview of Apoptosis.
Overview of apoptosis
•Programmed cell death.
•Apoptosis is a form of programmed cell death, or “cellular suicide.”
•Apoptosis is different from necrosis, in which cells die due to injury.
•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.
Why do cells undergo apoptosis?
- Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
- Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
- Apoptosis is part of development
- In many organisms, programmed cell death is a normal part of development.
The relationship between cancer cells and apoptosis.
Apoptosis can eliminate infected or cancerous cells.
When a cell’s DNA is damaged, it will typically detect the damage and try to repair it.
If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA.
When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.
However, “successful” cancer cells successfully evade the process of apoptosis.
This allows them to divide out of control and accumulate mutations (changes in their DNA).
Apoptosis is key to immune function
Apoptosis also plays an essential role in the development and maintenance of a healthy immune system.
Where are the weaknesses and symptoms of cancer cells?
The symptoms of cancer cells are in the nucleus.
The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.
DNA mutations also mutate in the nucleus.
Therefore, to treat cancer cells, we must first enter the nucleus.
Let the “regulatory cell gene” mechanism enter the nucleus to regulate
Are cancer cells aggressive?
After the action of Solamargine, the aggressiveness of cancer cells is alleviated.
So after using Solamargine, many patients feel that I am half better.
Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work).
Solamargine's major function mechanism:
When Solamargine enter,
Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.
Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.
Reduced drug resistance
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.
Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?
Solamargine vs cancer
Solamargine vs cancer
The picture shows the death of cancer cells.
The black and black parts are cancer cell nuclei.
Even if the nucleus ruptures, the cancer cells will die.
The figure shows that cancer cells can cause death.
The figure shows that cancer cells can cause death.
The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.
The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.
The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.
Best Chemotherapy Adjuvant. (1+1>487%)
Effectively improve chemotherapy effect and treatment.
ANTI-CANCER
Patent protection in 32 nations.
A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.
A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells.
SR-T100 combination therapy with effective result against breast cancer cells.
Combination Therapy | Research results for lung cancer cells.
A. Chemotherapy (100μM), 16% of cancer cell apoptosis.
B. Alone SM (4.8μM), 28% of cancer cell apoptosis.
C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.
D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.
SM has a clearing effect better than Chemotherapy.
The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.
SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).
SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times).
Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004.
Solamargine Q&A (English)
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Hypothalamic glioma | Visual pathway glioma | survival rate | Contents
Hypothalamic glioma | Visual pathway glioma | survival rate | Etymology and definitions
Hypothalamic glioma | Visual pathway glioma | survival rate | Signs and symptoms
Hypothalamic glioma | Visual pathway glioma | survival rate | Local symptoms
Hypothalamic glioma | Visual pathway glioma | survival rate | Systemic symptoms
Hypothalamic glioma | Visual pathway glioma | survival rate | Metastasis
Hypothalamic glioma | Visual pathway glioma | survival rate | Causes
Hypothalamic glioma | Visual pathway glioma | survival rate | Chemicals
Hypothalamic glioma | Visual pathway glioma | survival rate | Diet and exercise
Hypothalamic glioma | Visual pathway glioma | survival rate | Infection
Hypothalamic glioma | Visual pathway glioma | survival rate | Radiation
Hypothalamic glioma | Visual pathway glioma | survival rate | Heredity
Hypothalamic glioma | Visual pathway glioma | survival rate | Physical agents
Hypothalamic glioma | Visual pathway glioma | survival rate | Hormones
Hypothalamic glioma | Visual pathway glioma | survival rate | Autoimmune diseases
Hypothalamic glioma | Visual pathway glioma | survival rate | Pathophysiology
Hypothalamic glioma | Visual pathway glioma | survival rate | Genetics
Hypothalamic glioma | Visual pathway glioma | survival rate | Epigenetics
Hypothalamic glioma | Visual pathway glioma | survival rate | Metastasis
Hypothalamic glioma | Visual pathway glioma | survival rate | Metabolism
Hypothalamic glioma | Visual pathway glioma | survival rate | Diagnosis
Hypothalamic glioma | Visual pathway glioma | survival rate | Classification
Hypothalamic glioma | Visual pathway glioma | survival rate | Prevention
Hypothalamic glioma | Visual pathway glioma | survival rate | Dietary
Hypothalamic glioma | Visual pathway glioma | survival rate | Medication
Hypothalamic glioma | Visual pathway glioma | survival rate | Vaccination
Hypothalamic glioma | Visual pathway glioma | survival rate | Screening
Hypothalamic glioma | Visual pathway glioma | survival rate | Recommendations
Hypothalamic glioma | Visual pathway glioma | survival rate | Genetic testing
Hypothalamic glioma | Visual pathway glioma | survival rate | Management
Hypothalamic glioma | Visual pathway glioma | survival rate | Chemotherapy
Hypothalamic glioma | Visual pathway glioma | survival rate | Radiation
Hypothalamic glioma | Visual pathway glioma | survival rate | Surgery
Hypothalamic glioma | Visual pathway glioma | survival rate | Palliative care
Hypothalamic glioma | Visual pathway glioma | survival rate | Immunotherapy
Hypothalamic glioma | Visual pathway glioma | survival rate | Laser therapy
Hypothalamic glioma | Visual pathway glioma | survival rate | Alternative medicine
Hypothalamic glioma | Visual pathway glioma | survival rate | Prognosis
Hypothalamic glioma | Visual pathway glioma | survival rate | Epidemiology
Hypothalamic glioma | Visual pathway glioma | survival rate | History
Hypothalamic glioma | Visual pathway glioma | survival rate | Society and culture
Hypothalamic glioma | Visual pathway glioma | survival rate | Economic effect
Hypothalamic glioma | Visual pathway glioma | survival rate | Workplace
Hypothalamic glioma | Visual pathway glioma | survival rate | Research
Hypothalamic glioma | Visual pathway glioma | survival rate | Pregnancy
Hypothalamic glioma | Visual pathway glioma | survival rate | Other animals
Hypothalamic glioma | Visual pathway glioma | survival rate | Notes
Hypothalamic glioma | Visual pathway glioma | survival rate | Further reading
Hypothalamic glioma | Visual pathway glioma | survival rate | External links