Best adjuvant (assist) for chemotherapy | 1+1>487% |
Effectively improve chemotherapy effect, treatment, immunity.
Reduce side effects and recurrence.
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work |
Abstract / Summary / Overview of Apoptosis.
Why do cells undergo apoptosis?
The relationship between cancer cells and apoptosis.
Where are the weaknesses and symptoms of cancer cells?
Are cancer cells aggressive?
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
Solamargine vs cancer
Best Chemotherapy Adjuvant. (1+1>478%)
Effectively improve chemotherapy effect and cure.
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Extract : http://www.csh.org.tw/dr.tcj/educartion/f/web/Granulosa%20Theca%20cell%20tumors/index.htm
Granulosa-theca Cell Tumors
INTRODUCTION
Background: Three major types of ovarian neoplasms exist, with epithelial cell tumors (70%) comprising the largest group of tumors. Germ cell tumors occur less frequently (20%), while sex cord騯tromal tumors make up the smallest proportion, accounting for approximately 8% of all ovarian neoplasms.
Granulosa𩣱heca cell tumors, more commonly known as granulosa cell tumors (GCTs), belong to the sex cord騯tromal group and include tumors made up of granulosa cells, theca cells, and fibroblasts in varying degrees and combinations. Granulosa cell tumors account for approximately 2% of all ovarian tumors and can be divided into adult (95%) and juvenile (5%) types based on histologic findings.
Both subtypes are commonly estrogen producing, and it is the estrogen production that is often the reason for early diagnosis. However, while adult granulosa cell tumors usually occur in postmenopausal women and have late recurrences, most juvenile granulosa cell tumors occur before the age of 30 and recur within the first 3 years. Theca cell tumors almost always are benign and carry an excellent prognosis. The rare malignant thecoma likely represents a tumor with a small admixture of granulosa cells. For this reason, the remainder of the chapter focuses on granulosa cell tumors except where indicated.
Recognition of the signs and symptoms of abnormal hormone production and consideration of these tumors in the differential diagnosis of an adnexal mass can allow for early identification, timely surgical management, and excellent cure rates. Despite the good overall prognosis, long-term follow-up always is required in patients with granulosa cell tumors.
Abstract / Summary / Overview of Apoptosis.
Overview of apoptosis.
•Programmed cell death.
•Apoptosis is a form of programmed cell death, or “cellular suicide.”
•Apoptosis is different from necrosis, in which cells die due to injury.
•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.
Why do cells undergo apoptosis?
- Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
- Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
- Apoptosis is part of development
- In many organisms, programmed cell death is a normal part of development.
The relationship between cancer cells and apoptosis
Apoptosis can eliminate infected or cancerous cells.
When a cell’s DNA is damaged, it will typically detect the damage and try to repair it.
If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA.
When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.
However, “successful” cancer cells successfully evade the process of apoptosis.
This allows them to divide out of control and accumulate mutations (changes in their DNA).
Apoptosis is key to immune function.
Apoptosis also plays an essential role in the development and maintenance of a healthy immune system.
Where are the weaknesses and symptoms of cancer cells?
The symptoms of cancer cells are in the nucleus.
The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.
DNA mutations also mutate in the nucleus.
Therefore, to treat cancer cells, we must first enter the nucleus.
Let the “regulatory cell gene” mechanism enter the nucleus to regulate
Are cancer cells aggressive?
After the action of Solamargine, the aggressiveness of cancer cells is alleviated.
So after using Solamargine, many patients feel that I am half better.
Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work).
Solamargine's major function mechanism:
When Solamargine enter,
Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.
Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.
Reduced drug resistance.
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.
Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?
Solamargine vs cancer
Solamargine vs cancer
The picture shows the death of cancer cells.
The black and black parts are cancer cell nuclei.
Even if the nucleus ruptures, the cancer cells will die.
The figure shows that cancer cells can cause death.
The figure shows that cancer cells can cause death.
The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.
The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.
The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.
Best Chemotherapy Adjuvant. (1+1>487%)
Effectively improve chemotherapy effect and treatment.
ANTI-CANCER
Patent protection in 32 nations.
A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.
A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells.
SR-T100 combination therapy with effective result against breast cancer cells.
Combination Therapy | Research results for lung cancer cells.
A. Chemotherapy (100μM), 16% of cancer cell apoptosis.
B. Alone SM (4.8μM), 28% of cancer cell apoptosis.
C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.
D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.
SM has a clearing effect better than Chemotherapy.
The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.
SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).
SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times).
Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004.
Solamargine Q&A (English)
survival rate | cancer | Contents
survival rate | cancer | Etymology and definitions
survival rate | cancer | Signs and symptoms
survival rate | cancer | Local symptoms
survival rate | cancer | Systemic symptoms
survival rate | cancer | Metastasis
survival rate | cancer | Causes
survival rate | cancer | Chemicals
survival rate | cancer | Diet and exercise
survival rate | cancer | Infection
survival rate | cancer | Radiation
survival rate | cancer | Heredity
survival rate | cancer | Physical agents
survival rate | cancer | Hormones
survival rate | cancer | Autoimmune diseases
survival rate | cancer | Pathophysiology
survival rate | cancer | Genetics
survival rate | cancer | Epigenetics
survival rate | cancer | Metastasis
survival rate | cancer | Metabolism
survival rate | cancer | Diagnosis
survival rate | cancer | Classification
survival rate | cancer | Prevention
survival rate | cancer | Dietary
survival rate | cancer | Medication
survival rate | cancer | Vaccination
survival rate | cancer | Screening
survival rate | cancer | Recommendations
survival rate | cancer | Genetic testing
survival rate | cancer | Management
survival rate | cancer | Cancer
survival rate | cancer | Radiation
survival rate | cancer | Surgery
survival rate | cancer | Palliative care
survival rate | cancer | Immunotherapy
survival rate | cancer | Laser therapy
survival rate | cancer | Alternative medicine
survival rate | cancer | Prognosis
survival rate | cancer | Epidemiology
survival rate | cancer | History
survival rate | cancer | Society and culture
survival rate | cancer | Economic effect
survival rate | cancer | Workplace
survival rate | cancer | Research
survival rate | cancer | Pregnancy
survival rate | cancer | Other animals
survival rate | cancer | Notes
survival rate | cancer | Further reading
survival rate | cancer | External links
Granulosa cell tumor | survival rate | Contents
Granulosa cell tumor | survival rate | Etymology and definitions
Granulosa cell tumor | survival rate | Signs and symptoms
Granulosa cell tumor | survival rate | Local symptoms
Granulosa cell tumor | survival rate | Systemic symptoms
Granulosa cell tumor | survival rate | Metastasis
Granulosa cell tumor | survival rate | Causes
Granulosa cell tumor | survival rate | Chemicals
Granulosa cell tumor | survival rate | Diet and exercise
Granulosa cell tumor | survival rate | Infection
Granulosa cell tumor | survival rate | Radiation
Granulosa cell tumor | survival rate | Heredity
Granulosa cell tumor | survival rate | Physical agents
Granulosa cell tumor | survival rate | Hormones
Granulosa cell tumor | survival rate | Autoimmune diseases
Granulosa cell tumor | survival rate | Pathophysiology
Granulosa cell tumor | survival rate | Genetics
Granulosa cell tumor | survival rate | Epigenetics
Granulosa cell tumor | survival rate | Metastasis
Granulosa cell tumor | survival rate | Metabolism
Granulosa cell tumor | survival rate | Diagnosis
Granulosa cell tumor | survival rate | Classification
Granulosa cell tumor | survival rate | Prevention
Granulosa cell tumor | survival rate | Dietary
Granulosa cell tumor | survival rate | Medication
Granulosa cell tumor | survival rate | Vaccination
Granulosa cell tumor | survival rate | Screening
Granulosa cell tumor | survival rate | Recommendations
Granulosa cell tumor | survival rate | Genetic testing
Granulosa cell tumor | survival rate | Management
Granulosa cell tumor | survival rate | Chemotherapy
Granulosa cell tumor | survival rate | Radiation
Granulosa cell tumor | survival rate | Surgery
Granulosa cell tumor | survival rate | Palliative care
Granulosa cell tumor | survival rate | Immunotherapy
Granulosa cell tumor | survival rate | Laser therapy
Granulosa cell tumor | survival rate | Alternative medicine
Granulosa cell tumor | survival rate | Prognosis
Granulosa cell tumor | survival rate | Epidemiology
Granulosa cell tumor | survival rate | History
Granulosa cell tumor | survival rate | Society and culture
Granulosa cell tumor | survival rate | Economic effect
Granulosa cell tumor | survival rate | Workplace
Granulosa cell tumor | survival rate | Research
Granulosa cell tumor | survival rate | Pregnancy
Granulosa cell tumor | survival rate | Other animals
Granulosa cell tumor | survival rate | Notes
Granulosa cell tumor | survival rate | Further reading
Granulosa cell tumor | survival rate | External links
