Best adjuvant (assist) for chemotherapy. | 1+1>487%
Effectively improve chemotherapy effect, treatment, immunity.
Reduce side effects and recurrence.
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work |
Abstract / Summary / Overview of Apoptosis.
Why do cells undergo apoptosis?
The relationship between cancer cells and apoptosis.
Where are the weaknesses and symptoms of cancer cells?
Are cancer cells aggressive?
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
Solamargine vs cancer
Best Chemotherapy Adjuvant. (1+1>478%)
Effectively improve chemotherapy effect and cure.
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Extract : https://www.cancer.gov/about-cancer/understanding/what-is-cancer
The Definition of Cancer
Cancer is a disease in which some of the body’s cells grow uncontrollably and spread to other parts of the body.
Cancer can start almost anywhere in the human body, which is made up of trillions of cells. Normally, human cells grow and multiply (through a process called cell division) to form new cells as the body needs them. When cells grow old or become damaged, they die, and new cells take their place.
Sometimes this orderly process breaks down, and abnormal or damaged cells grow and multiply when they shouldn’t. These cells may form tumors, which are lumps of tissue. Tumors can be cancerous or not cancerous (benign).
Cancerous tumors spread into, or invade, nearby tissues and can travel to distant places in the body to form new tumors (a process called metastasis). Cancerous tumors may also be called malignant tumors. Many cancers form solid tumors, but cancers of the blood, such as leukemias, generally do not.
Benign tumors do not spread into, or invade, nearby tissues. When removed, benign tumors usually don’t grow back, whereas cancerous tumors sometimes do. Benign tumors can sometimes be quite large, however. Some can cause serious symptoms or be life threatening, such as benign tumors in the brain.
Abstract / Summary / Overview of Apoptosis.
Overview of apoptosis
•Programmed cell death
•Apoptosis is a form of programmed cell death, or “cellular suicide.”
•Apoptosis is different from necrosis, in which cells die due to injury.
•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.
Why do cells undergo apoptosis?
- Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
- Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
- Apoptosis is part of development
- In many organisms, programmed cell death is a normal part of development.
The relationship between cancer cells and apoptosis
Apoptosis can eliminate infected or cancerous cells.
When a cell’s DNA is damaged, it will typically detect the damage and try to repair it.
If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA.
When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.
However, “successful” cancer cells successfully evade the process of apoptosis.
This allows them to divide out of control and accumulate mutations (changes in their DNA).
Apoptosis is key to immune function
Apoptosis also plays an essential role in the development and maintenance of a healthy immune system.
Where are the weaknesses and symptoms of cancer cells?
The symptoms of cancer cells are in the nucleus.
The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.
DNA mutations also mutate in the nucleus.
Therefore, to treat cancer cells, we must first enter the nucleus.
Let the “regulatory cell gene” mechanism enter the nucleus to regulate
Are cancer cells aggressive?
After the action of Solamargine, the aggressiveness of cancer cells is alleviated.
So after using Solamargine, many patients feel that I am half better.
Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work).
Solamargine's major function mechanism:
When Solamargine enter,
Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.
Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.
Reduced drug resistance
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.
Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?
Solamargine vs cancer
Solamargine vs cancer
The picture shows the death of cancer cells.
The black and black parts are cancer cell nuclei.
Even if the nucleus ruptures, the cancer cells will die.
The figure shows that cancer cells can cause death.
The figure shows that cancer cells can cause death.
The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.
The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.
The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.
Best Chemotherapy Adjuvant. (1+1>487%)
Effectively improve chemotherapy effect and treatment.
ANTI-CANCER
Patent protection in 32 nations.
A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.
A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells.
SR-T100 combination therapy with effective result against breast cancer cells.
Combination Therapy | Research results for lung cancer cells.
A. Chemotherapy (100μM), 16% of cancer cell apoptosis.
B. Alone SM (4.8μM), 28% of cancer cell apoptosis.
C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.
D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.
SM has a clearing effect better than Chemotherapy.
The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.
SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).
SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times).
Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004.
Solamargine Q&A (English)
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Cancer | stage 1 | stage 2 | survival rate | Contents
Cancer | stage 1 | stage 2 | survival rate | Etymology and definitions
Cancer | stage 1 | stage 2 | survival rate | Signs and symptoms
Cancer | stage 1 | stage 2 | survival rate | Local symptoms
Cancer | stage 1 | stage 2 | survival rate | Systemic symptoms
Cancer | stage 1 | stage 2 | survival rate | Metastasis
Cancer | stage 1 | stage 2 | survival rate | Causes
Cancer | stage 1 | stage 2 | survival rate | Chemicals
Cancer | stage 1 | stage 2 | survival rate | Diet and exercise
Cancer | stage 1 | stage 2 | survival rate | Infection
Cancer | stage 1 | stage 2 | survival rate | Radiation
Cancer | stage 1 | stage 2 | survival rate | Heredity
Cancer | stage 1 | stage 2 | survival rate | Physical agents
Cancer | stage 1 | stage 2 | survival rate | Hormones
Cancer | stage 1 | stage 2 | survival rate | Autoimmune diseases
Cancer | stage 1 | stage 2 | survival rate | Pathophysiology
Cancer | stage 1 | stage 2 | survival rate | Genetics
Cancer | stage 1 | stage 2 | survival rate | Epigenetics
Cancer | stage 1 | stage 2 | survival rate | Metastasis
Cancer | stage 1 | stage 2 | survival rate | Metabolism
Cancer | stage 1 | stage 2 | survival rate | Diagnosis
Cancer | stage 1 | stage 2 | survival rate | Classification
Cancer | stage 1 | stage 2 | survival rate | Prevention
Cancer | stage 1 | stage 2 | survival rate | Dietary
Cancer | stage 1 | stage 2 | survival rate | Medication
Cancer | stage 1 | stage 2 | survival rate | Vaccination
Cancer | stage 1 | stage 2 | survival rate | Screening
Cancer | stage 1 | stage 2 | survival rate | Recommendations
Cancer | stage 1 | stage 2 | survival rate | Genetic testing
Cancer | stage 1 | stage 2 | survival rate | Management
Cancer | stage 1 | stage 2 | survival rate | Chemotherapy
Cancer | stage 1 | stage 2 | survival rate | Radiation
Cancer | stage 1 | stage 2 | survival rate | Surgery
Cancer | stage 1 | stage 2 | survival rate | Palliative care
Cancer | stage 1 | stage 2 | survival rate | Immunotherapy
Cancer | stage 1 | stage 2 | survival rate | Laser therapy
Cancer | stage 1 | stage 2 | survival rate | Alternative medicine
Cancer | stage 1 | stage 2 | survival rate | Prognosis
Cancer | stage 1 | stage 2 | survival rate | Epidemiology
Cancer | stage 1 | stage 2 | survival rate | History
Cancer | stage 1 | stage 2 | survival rate | Society and culture
Cancer | stage 1 | stage 2 | survival rate | Economic effect
Cancer | stage 1 | stage 2 | survival rate | Workplace
Cancer | stage 1 | stage 2 | survival rate | Research
Cancer | stage 1 | stage 2 | survival rate | Pregnancy
Cancer | stage 1 | stage 2 | survival rate | Other animals
Cancer | stage 1 | stage 2 | survival rate | Notes
Cancer | stage 1 | stage 2 | survival rate | Further reading
Cancer | stage 1 | stage 2 | survival rate | External links