Best adjuvant (assist) for chemotherapy | 1+1>487% |
Effectively improve chemotherapy effect, treatment, immunity |
Reduce side effects and recurrence |
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work |
Abstract / Summary / Overview of Apoptosis
Why do cells undergo apoptosis?
The relationship between cancer cells and apoptosis
Where are the weaknesses and symptoms of cancer cells?
Are cancer cells aggressive?
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
Solamargine vs cancer
Best Chemotherapy Adjuvant (1+1>478%) Effectively improve chemotherapy effect and cure
Best adjuvant (assist) for chemotherapy | 1+1>487%
Effectively improve chemotherapy effect, treatment, immunity
Reduce side effects and recurrence
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work
Abstract / Summary / Overview of Apoptosis
Overview of apoptosis
•Programmed cell death
•Apoptosis is a form of programmed cell death, or “cellular suicide.”
•Apoptosis is different from necrosis, in which cells die due to injury.
•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.
Why do cells undergo apoptosis?
- Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
- Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
- Apoptosis is part of development
- In many organisms, programmed cell death is a normal part of development.
The relationship between cancer cells and apoptosis
Apoptosis can eliminate infected or cancerous cells.
When a cell’s DNA is damaged, it will typically detect the damage and try to repair it.
If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA.
When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.
However, “successful” cancer cells successfully evade the process of apoptosis.
This allows them to divide out of control and accumulate mutations (changes in their DNA).
Apoptosis is key to immune function
Apoptosis also plays an essential role in the development and maintenance of a healthy immune system.
Where are the weaknesses and symptoms of cancer cells?
The symptoms of cancer cells are in the nucleus.
The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.
DNA mutations also mutate in the nucleus.
Therefore, to treat cancer cells, we must first enter the nucleus.
Let the “regulatory cell gene” mechanism enter the nucleus to regulate
Are cancer cells aggressive?
After the action of Solamargine, the aggressiveness of cancer cells is alleviated.
So after using Solamargine, many patients feel that I am half better.
Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
When Solamargine enter,
Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.
Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.
Reduced drug resistance
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.
Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?
Solamargine vs cancer
Solamargine vs cancer
The picture shows the death of cancer cells.
The black and black parts are cancer cell nuclei.
Even if the nucleus ruptures, the cancer cells will die.
The figure shows that cancer cells can cause death.
The figure shows that cancer cells can cause death.
The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.
The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.
The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.
Best Chemotherapy Adjuvant (1+1>487%) Effectively improve chemotherapy effect and treatment
ANTI-CANCER
Patent protection in 32 nations.
A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.
A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells
SR-T100 combination therapy with effective result against breast cancer cells.
Combination Therapy | Research results for lung cancer cells.
A. Chemotherapy (100μM), 16% of cancer cell apoptosis.
B. Alone SM (4.8μM), 28% of cancer cell apoptosis.
C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.
D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.
SM has a clearing effect better than Chemotherapy.
The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.
SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).
SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times)
Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004
Solamargine Q&A (English)
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위장 기질 종양 | GIST | 생존율 | Contents
위장 기질 종양 | GIST | 생존율 | Etymology and definitions
위장 기질 종양 | GIST | 생존율 | Signs and symptoms
위장 기질 종양 | GIST | 생존율 | Local symptoms
위장 기질 종양 | GIST | 생존율 | Systemic symptoms
위장 기질 종양 | GIST | 생존율 | Metastasis
위장 기질 종양 | GIST | 생존율 | Causes
위장 기질 종양 | GIST | 생존율 | Chemicals
위장 기질 종양 | GIST | 생존율 | Diet and exercise
위장 기질 종양 | GIST | 생존율 | Infection
위장 기질 종양 | GIST | 생존율 | Radiation
위장 기질 종양 | GIST | 생존율 | Heredity
위장 기질 종양 | GIST | 생존율 | Physical agents
위장 기질 종양 | GIST | 생존율 | Hormones
위장 기질 종양 | GIST | 생존율 | Autoimmune diseases
위장 기질 종양 | GIST | 생존율 | Pathophysiology
위장 기질 종양 | GIST | 생존율 | Genetics
위장 기질 종양 | GIST | 생존율 | Epigenetics
위장 기질 종양 | GIST | 생존율 | Metastasis
위장 기질 종양 | GIST | 생존율 | Metabolism
위장 기질 종양 | GIST | 생존율 | Diagnosis
위장 기질 종양 | GIST | 생존율 | Classification
위장 기질 종양 | GIST | 생존율 | Prevention
위장 기질 종양 | GIST | 생존율 | Dietary
위장 기질 종양 | GIST | 생존율 | Medication
위장 기질 종양 | GIST | 생존율 | Vaccination
위장 기질 종양 | GIST | 생존율 | Screening
위장 기질 종양 | GIST | 생존율 | Recommendations
위장 기질 종양 | GIST | 생존율 | Genetic testing
위장 기질 종양 | GIST | 생존율 | Management
위장 기질 종양 | GIST | 생존율 | Chemotherapy
위장 기질 종양 | GIST | 생존율 | Radiation
위장 기질 종양 | GIST | 생존율 | Surgery
위장 기질 종양 | GIST | 생존율 | Palliative care
위장 기질 종양 | GIST | 생존율 | Immunotherapy
위장 기질 종양 | GIST | 생존율 | Laser therapy
위장 기질 종양 | GIST | 생존율 | Alternative medicine
위장 기질 종양 | GIST | 생존율 | Prognosis
위장 기질 종양 | GIST | 생존율 | Epidemiology
위장 기질 종양 | GIST | 생존율 | History
위장 기질 종양 | GIST | 생존율 | Society and culture
위장 기질 종양 | GIST | 생존율 | Economic effect
위장 기질 종양 | GIST | 생존율 | Workplace
위장 기질 종양 | GIST | 생존율 | Research
위장 기질 종양 | GIST | 생존율 | Pregnancy
위장 기질 종양 | GIST | 생존율 | Other animals
위장 기질 종양 | GIST | 생존율 | Notes
위장 기질 종양 | GIST | 생존율 | Further reading
위장 기질 종양 | GIST | 생존율 | External links