Best adjuvant (assist) for chemotherapy | 1+1>487% |
Effectively improve chemotherapy effect, treatment, immunity |
Reduce side effects and recurrence |
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work |
Abstract / Summary / Overview of Apoptosis
Why do cells undergo apoptosis?
The relationship between cancer cells and apoptosis
Where are the weaknesses and symptoms of cancer cells?
Are cancer cells aggressive?
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
Solamargine vs cancer
Best Chemotherapy Adjuvant (1+1>478%) Effectively improve chemotherapy effect and cure
Best adjuvant (assist) for chemotherapy | 1+1>487%
Effectively improve chemotherapy effect, treatment, immunity
Reduce side effects and recurrence
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work
Abstract / Summary / Overview of Apoptosis
![Apoptosis.jpg Apoptosis.jpg](https://cdn.store-assets.com/s/276434/f/6301875.png)
Overview of apoptosis
•Programmed cell death
•Apoptosis is a form of programmed cell death, or “cellular suicide.”
•Apoptosis is different from necrosis, in which cells die due to injury.
•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.
Why do cells undergo apoptosis?
- Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
- Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
- Apoptosis is part of development
- In many organisms, programmed cell death is a normal part of development.
The relationship between cancer cells and apoptosis
Apoptosis can eliminate infected or cancerous cells.
When a cell’s DNA is damaged, it will typically detect the damage and try to repair it.
If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA.
When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.
However, “successful” cancer cells successfully evade the process of apoptosis.
This allows them to divide out of control and accumulate mutations (changes in their DNA).
Apoptosis is key to immune function
Apoptosis also plays an essential role in the development and maintenance of a healthy immune system.
Where are the weaknesses and symptoms of cancer cells?
The symptoms of cancer cells are in the nucleus.
The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.
DNA mutations also mutate in the nucleus.
Therefore, to treat cancer cells, we must first enter the nucleus.
Let the “regulatory cell gene” mechanism enter the nucleus to regulate
Are cancer cells aggressive?
After the action of Solamargine, the aggressiveness of cancer cells is alleviated.
So after using Solamargine, many patients feel that I am half better.
Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
![sr-t100_apoptosis_mechanism005.jpg sr-t100_apoptosis_mechanism005.jpg](https://cdn.store-assets.com/s/276434/f/6301874.png)
Solamargine's major function mechanism:
When Solamargine enter,
Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.
Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.
Reduced drug resistance
When cancer cells are less resistant to drugs, chemotherapy becomes more effective.
Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.
Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?
![Chemotherapy_01.jpg Chemotherapy_01.jpg](https://cdn.store-assets.com/s/276434/f/6301865.png)
Solamargine vs cancer
![cell apoptosis.jpg cell apoptosis.jpg](https://cdn.store-assets.com/s/276434/f/6301872.png)
Solamargine vs cancer
The picture shows the death of cancer cells.
The black and black parts are cancer cell nuclei.
Even if the nucleus ruptures, the cancer cells will die.
The figure shows that cancer cells can cause death.
![cancer cell apoptosis_01_800.jpg cancer cell apoptosis_01_800.jpg](https://cdn.store-assets.com/s/276434/f/6301869.png)
The figure shows that cancer cells can cause death.
The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.
The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.
The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.
Best Chemotherapy Adjuvant (1+1>487%) Effectively improve chemotherapy effect and treatment![solamargine vs cancer_lung cancer cell.jpg solamargine vs cancer_lung cancer cell.jpg](https://cdn.store-assets.com/s/276434/f/6301870.png)
ANTI-CANCER
Patent protection in 32 nations.
A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.
![solamargine vs cancer_breast cancer cell_01_800.jpg solamargine vs cancer_breast cancer cell_01_800.jpg](https://cdn.store-assets.com/s/276434/f/6301868.png)
A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells
![solamargine combined treatment therapy_01_800.jpg solamargine combined treatment therapy_01_800.jpg](https://cdn.store-assets.com/s/276434/f/6301871.png)
SR-T100 combination therapy with effective result against breast cancer cells.
![solamargine combined treatment therapy_03R12_800.jpg solamargine combined treatment therapy_03R12_800.jpg](https://cdn.store-assets.com/s/276434/f/6301873.png)
Combination Therapy | Research results for lung cancer cells.
A. Chemotherapy (100μM), 16% of cancer cell apoptosis.
B. Alone SM (4.8μM), 28% of cancer cell apoptosis.
C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.
D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.
SM has a clearing effect better than Chemotherapy.
The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.
SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).
SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times)
Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004
![justnow_02.jpg justnow_02.jpg](https://cdn.store-assets.com/s/276434/f/6301867.png)
Solamargine Q&A (English)
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survival rate | cancer | Etymology and definitions
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Ailse próstatach | Ráta marthanais | Contents
Ailse próstatach | Ráta marthanais | Etymology and definitions
Ailse próstatach | Ráta marthanais | Signs and symptoms
Ailse próstatach | Ráta marthanais | Local symptoms
Ailse próstatach | Ráta marthanais | Systemic symptoms
Ailse próstatach | Ráta marthanais | Metastasis
Ailse próstatach | Ráta marthanais | Causes
Ailse próstatach | Ráta marthanais | Chemicals
Ailse próstatach | Ráta marthanais | Diet and exercise
Ailse próstatach | Ráta marthanais | Infection
Ailse próstatach | Ráta marthanais | Radiation
Ailse próstatach | Ráta marthanais | Heredity
Ailse próstatach | Ráta marthanais | Physical agents
Ailse próstatach | Ráta marthanais | Hormones
Ailse próstatach | Ráta marthanais | Autoimmune diseases
Ailse próstatach | Ráta marthanais | Pathophysiology
Ailse próstatach | Ráta marthanais | Genetics
Ailse próstatach | Ráta marthanais | Epigenetics
Ailse próstatach | Ráta marthanais | Metastasis
Ailse próstatach | Ráta marthanais | Metabolism
Ailse próstatach | Ráta marthanais | Diagnosis
Ailse próstatach | Ráta marthanais | Classification
Ailse próstatach | Ráta marthanais | Prevention
Ailse próstatach | Ráta marthanais | Dietary
Ailse próstatach | Ráta marthanais | Medication
Ailse próstatach | Ráta marthanais | Vaccination
Ailse próstatach | Ráta marthanais | Screening
Ailse próstatach | Ráta marthanais | Recommendations
Ailse próstatach | Ráta marthanais | Genetic testing
Ailse próstatach | Ráta marthanais | Management
Ailse próstatach | Ráta marthanais | Chemotherapy
Ailse próstatach | Ráta marthanais | Radiation
Ailse próstatach | Ráta marthanais | Surgery
Ailse próstatach | Ráta marthanais | Palliative care
Ailse próstatach | Ráta marthanais | Immunotherapy
Ailse próstatach | Ráta marthanais | Laser therapy
Ailse próstatach | Ráta marthanais | Alternative medicine
Ailse próstatach | Ráta marthanais | Prognosis
Ailse próstatach | Ráta marthanais | Epidemiology
Ailse próstatach | Ráta marthanais | History
Ailse próstatach | Ráta marthanais | Society and culture
Ailse próstatach | Ráta marthanais | Economic effect
Ailse próstatach | Ráta marthanais | Workplace
Ailse próstatach | Ráta marthanais | Research
Ailse próstatach | Ráta marthanais | Pregnancy
Ailse próstatach | Ráta marthanais | Other animals
Ailse próstatach | Ráta marthanais | Notes
Ailse próstatach | Ráta marthanais | Further reading
Ailse próstatach | Ráta marthanais | External links